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Posts by Nicholas Myers

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Since Hrvoje hasn't been active on the forum for a while, I’ll attempt to answer these questions myself. Just a disclaimer: I don't practice gastroenterology anymore, so there's a margin for error here.
Angela Wright said:Is it possible that the sludge seen on the ultrasound cleared out, or would it be invisible on an MRI?

An MRCP can certainly detect gallbladder sludge; in fact, it might even be more sensitive than an ultrasound. Source for this.

Furthermore, gallbladder sludge is quite common toward the end of pregnancy and for some time postpartum, though in most cases, it resolves on its own. Source for this.

It is entirely possible that the sludge simply cleared out in this instance.
So, what am I looking at now with a folded gallbladder? Is it possible the folding was caused by those two subsequent attacks following the first ultrasound?

Based on the medical literature—and my own training—a folded gallbladder is considered an anatomical variation present from embryonic development. It's unlikely to be caused by body positioning or a baby kicking near the right rib cage.

Generally, a folded gallbladder isn't noted as a specific clinical issue. From what I understand, the gastroenterologists I've consulted suggest looking elsewhere for the cause of symptoms rather than the fold itself. That said, if certain types of folding carry specific significance, that's a question for someone with subspecialty expertise.

Gallbladder sludge can mask small stones during an ultrasound, so it's possible tiny stones were missed and were actually the culprit. These micro-stones (microlithiasis) can definitely cause issues.

The general consensus is that surgery is reserved for symptomatic patients—those actually experiencing trouble.

Also, looking at the referral, the gastroenterologist noted "suspected liver lesions and microlithiasis." I wouldn't assume those two findings are necessarily linked.
edit: I didn't do the liver function tests because they told me to wait until a few days before my follow-up (I have a standing lab order), so I'm not sure where things stand. But the MRI shows the liver is fine. Should I head to the local clinic to get the bloodwork done anyway?

That depends on when your appointment is. Assuming the doctor will see you relatively soon, waiting a week or two shouldn't make much difference.
reddrifter11 said:Google can tell you just about anything, which is why I’m asking here. Is there any reason to worry? Thanks.

The results are essentially normal. Personally, I don't see much cause for concern regarding those slightly low TIBC levels.

Best, 🙂
Michelle Harris27 said:I just got my cervical results back...

If anyone can tell me what to expect...

Ureaplasma >10 (on a 5 scale)
Mycoplasma was not isolated
Cervical swab - bacterial aerobic and microbiological - normal vaginal flora

PCR for C. trachomatis - Roche PCR COBAS TaqMan CT test: Chlamydia trachomatis DNA not detected

Ma'am,

The report likely includes an antibiogram—essentially a list showing which antibiotics the ureaplasma is sensitive to. Since the ureaplasma count is quite high here, antibiotic treatment is necessary. It isn't always straightforward, though; these bacteria are often resistant to several drugs and typically only respond to a few, such as azithromycin or doxycycline. Your partner definitely needs to be tested and treated as well if required. Finally, you’ll need a follow-up swab to confirm the treatment worked (but don't do this until at least two weeks after finishing the medication).
Kimberly Bailey6 said:Hi, could someone please help me interpret these results:

Day 2 of cycle:
Estradiol: 326 (110-1100 pmol/L)
Progesterone: 8.2 (follicular phase: up to 3.2 nmol/L)
LH: 3.1 (3-12 IU/L)
FSH: 3.9 (2-12 IU/L)
Prolactin: 15.8 (4-23 µg/L)
Total Testosterone: 1.4 (0.2-2.6 nmol/L)
Free Testosterone: 23.8 (3.5-30 pmol/L)
SHBG: 35 (26.1-110 nmol/L)
DHEA-S: 4.28 (2.68-9.23 µmol/L)
Androstenedione: 13.10 (1.0-11.5 nmol/L)
ACTH: 4.9 (1.6-13.9 pmol/L)
AMH: 5.96 (1.3-7.0 ng/ml)

Day 22 of cycle:
Progesterone: 9.6 (luteal phase should be 20+ nmol/L)

T3, T4, and TSH are fine.

Testing due to anovulation and PCOS. My cycles have always been irregular (anywhere from 35 days to 10 months).

I can't find any info on why progesterone might be elevated during the first phase?
Could the high androstenedione be causing the anovulation?

Elevated androstenedione levels can be linked to PCOS; in fact, they are frequently seen in those cases. One possible explanation involves insulin resistance—higher insulin levels can trigger the ovaries to produce more androstenedione.

Androstenedione itself can also originate from the adrenal glands.

It would be wise to discuss further testing with the gynecologist who ordered these, such as checking other hormones (like cortisol or 17-OHP) or investigating insulin resistance via an OGTT. Since this falls outside my specific area of expertise, I can't offer a definitive opinion on those matters.

Low progesterone during the second phase of the cycle supports the diagnosis of anovulation, but as for why it might be elevated in the first phase... well, that's a much harder question to answer.

Best regards. 🙂
Starfish, Angela Wright, and boldnomad45, I've moved your posts to the thread Urination - pain, burning, and other issues within the Women's health PDF section.
Need help finding a hematologist!!!! in Health ·
J&B and Ivanicapeca, I've moved your posts over to the INFO DESK thread.

I'm locking this thread down—it’s just not going anywhere.
Olivia Johnson83 said:The test was ordered because the capillaroscopy showed an atypical scleroderma pattern. There’s suspicion of scleroderma.

It looks like you might have positive CENP-B antibodies—it's worth double-checking the reference ranges on your lab report. If those are present, combined with your clinical presentation, it could point toward scleroderma or a risk of developing it down the road. Usually, this manifests as a limited form that progresses slowly and stays confined to the skin.
The data doesn't lie. If you want clarity, you have to look at the numbers, not the noise. Everything boils down to the specifics. kaže:
Results from a week ago (March 5th): ALP 845 U/L.
Today’s results (March 12): ALP 510 U/L.

Reference ranges:
Men: 60 – 142 U/L

To whom it may concern,

It’s difficult to pinpoint exactly what's driving that ALP spike in your case. It could be an underlying issue—something like mono or another viral infection. Theoretically, azithromycin can have hepatotoxic effects that bump up both liver enzymes and ALP, though proving that connection would be a massive uphill battle. Besides, you haven't shared the rest of your labs; you've focused solely on the ALP, and looking at isolated numbers in a vacuum doesn't tell us much.

It’s certainly a positive sign that ALP is trending down, but I’d suggest getting a specialist to weigh in and provide a formal evaluation.

Greetings. 🙂
I don't know what you're trying to say, but let's get back to the point. kaže:
I'm looking for a professional opinion...

Is it possible for someone who completed the full hepatitis B vaccine series—three doses over six months—to still test negative for anti-HBs?
The vaccination rollout kicked off in late 2011.

It’s entirely possible that someone failed to develop any protective antibodies—essentially, the vaccination didn't take. We need to dig deeper and figure out exactly why that happened.

Olivia Johnson83 said:Can someone help me make sense of these lab results? S(ANA) (ENA) IIF centromere titer < 1:100.
dsDNA negative. IU/ml < 30. Negative.
30-40. On the edge.
Positive results for over 40.
SS-A 52 negative, at 52 AU/ml.
SS-A 60 negative at 60 AU/ml.
SS-B negative. Results are back.
Serum Cortisol levels are coming back negative at -ll- AU/ml.
(S) Sm/RNP negative. AU/ml -ll-
(S) DNA-topo 1 negative AU/ml -ll-
(S) Jo-1 negative AU/ml -ll-
(S) CENP-B 300 * AU/ml -ll-
(S) RIBO negative.

What was the reason for this test?
Gerald Thompson11 said:AGLM and all that, but I don't see a clear link between high AGLM and anti-TPO... having high anti-TPO doesn't feel right.

Hello,

The connection lies in the fact that both results can point toward autoimmune conditions—essentially, scenarios where the body attacks itself. It’s possible you're dealing with an autoimmune thyroid issue. You'll need more testing; specifically, checking your thyroid hormone levels, and perhaps getting a thyroid ultrasound.

As for the AGLM, that could suggest an autoimmune liver issue, which would require further investigation.

Best regards. 🙂
Wrist pain from mouse usage? in Health ·
Ladies and gentlemen, please respect the guidelines for the Health subforum. Refrain from discussing alternative therapies here, such as bone-setting. Previous comments have been removed.
Donna Garcia52 said:Should I be worried about elevated iron levels that I’ve never seen before? Should I go looking for more tests right away, or just wait and retest in a while?
My total cholesterol is high. However, compared to my other cholesterol numbers, how much of a dietary shift is actually required? Is this level concerning? I eat fairly healthy and stay active.

Thanks in advance for any insight!

Hello,

Iron concentration is quite variable. Ferritin is a much more reliable indicator of your actual iron stores, so I wouldn't lose sleep over an iron reading sitting at the upper limit. No extra testing seems necessary right now.

Regarding cholesterol, the context matters: age, blood pressure, smoking status, weight, waist-to-hip ratio, family history, and whether you have diabetes.

Values like yours can often be managed effectively through dietary adjustments and increased physical activity. They aren't an immediate cause for alarm, but if you're young, it's worth thinking about long-term vascular health. It's better to get those numbers under control now.

Best regards. 🙂
crimsonmoose2 said:My assumption was that SKORT referred to serum Cortisol, drawn directly from the blood. When I asked my nurse how the testing process worked—specifically alongside the TSH, T3, and T4 panels—she confirmed that was exactly what it meant.

No, they only draw my blood at the lab. They hand me the results for those last three hormones right there on the spot, but for the SKORT, there’s just a note stating that the 24-hour urine Cortisol wasn't submitted for the SKORT testing.

So, the question remains: when testing for SKORT, are we looking at blood work or a urine sample?
A quick search online shows that abbreviation is virtually nonexistent. When looking for cortisol testing, you really only have two practical options: serum Cortisol or urine Cortisol.

Could someone please clarify the difference here if you have the expertise? I’m trying to figure out who actually missed the mark in this discussion.

To whom it may concern,

No, that abbreviation actually refers to free cortisol levels, which are measured via a 24-hour urine Cortisol test. You really should have clarified that with whoever ordered the lab work in the first place.

Greetings. 🙂

dustygardener11 said:Could someone please help me interpret these blood test results?

EBV VCA IgG came back positive at 2.35 (reference range EBV VCA IgM came back negative.
EBV EBNA IgG came back positive at 7.65 (reference range CMV IgG came back positive at 29 (reference range The CMV IgM came back negative.

Am I dealing with mono right now, or is this just something I already had in the past making a comeback?

To whom it may concern,

We’re still missing the EA marker data to be truly precise, but based on this specific constellation of findings, I wouldn't point toward an acute or active case of mononucleosis.

Greetings. 🙂

Betty Davis73 said:Hello.

Could someone please help me interpret these results?

http://www.picamatic.com/view/10181692_20140303_102114/

I'm six months pregnant. This test was just one small part of a massive battery of labs I had done.

I'm particularly curious about what that DZ 152 note under the comments actually signifies.

Much appreciated! 🙂

To whom it may concern,

Just a heads-up regarding that "DZ 152" note: it’s strictly an internal shorthand for lab staff. It has zero bearing on your actual results. What the data actually shows is that you carry antibodies for both types of herpes simplex. Essentially, you've been exposed to both viruses at some point in the past, though the test can't pinpoint exactly when that happened.

Best regards. 🙂
Changes or growths on eyelids? in Health ·
Michelle Bishop44 said:What’s the standard procedure for removing syringomas?

Options include surgical excision, laser therapy, or electrocautery. It really boils down to the doctor's assessment and what the patient decides is best.
Please refrain from starting duplicate threads or cross-posting the same message across multiple discussions; it violates our community guidelines. A single inquiry under the "glasses" thread is sufficient, even if the topic might technically fit better under Consumer Law regarding consumer protection rights.
Conphas, I've moved your post over to the thread Heart and lung scans - findings and opinions.
Anthony Davis72 said:Thanks for the input. I checked with a local clinic and they have about 15 different markers listed for hepatitis. Should I be testing for everything, or can we narrow this down to just a few specific ones?

Given that your liver enzyme levels are off, you should be able to get those viral hepatitis markers covered by your insurance via a standard referral. There’s really no reason to pay out of pocket for this.
ferallynx63 said:Hi there,

I'm dealing with some bladder issues and have to go in for urine and blood work next week. Is there any chance they'll pick up on THC levels during those tests? I don't think drug screening is part of this, especially since the focus is on the bladder, not narcotics.

It isn't included.
Angela Wright said:If you're taking Moduretic, you absolutely have to take Kalinor with it. Otherwise, your electrolyte levels will tank completely.

Angela, I have to correct that statement.

Moduretic is actually a potassium-sparing diuretic. Generally speaking, you wouldn't be advised to take potassium supplements like Kalinor or potassium chloride alongside it. There are extremely rare exceptions—cases involving severe hypokalemia—but even then, it requires strict medical supervision and constant electrolyte monitoring.

Otherwise, no.
Carl Ramos5 said:Thanks for getting back to me. Could you tell me a bit more about what R74 and M54 actually mean? Thanks.🙂 Best regards.

I covered R74 previously, so I'll just pull my previous response here:
Maria Fisher46 said:Hello,

Your results show elevated liver enzymes. These levels are only mildly increased—for context, medical guidelines often categorize anything under 250 IU as mild, 250 to 1000 IU as moderate, and anything over 1000 IU as severe. This isn't an emergency or a reason to panic, but it is a clear signal to follow up with your primary care physician. They will likely suggest some lifestyle adjustments followed by a repeat liver function test in about 4 to 6 weeks.

Essentially, this indicates some level of liver cell damage. However, these numbers alone don't pinpoint the specific cause.

Any condition causing moderate inflammation or cellular damage in the liver can lead to a slight rise in aminotransferases, similar to what you're seeing.

Factors like an unhealthy lifestyle—excessive alcohol consumption, poor diet, or obesity—could be responsible, though there are many potential culprits.

If the issue stems from alcohol or diet, one would expect the levels to normalize once those habits are corrected.

The best course of action is to adopt a liver-friendly diet (which means cutting out alcohol, avoiding fatty, sugary, fried, or processed foods), working on weight management, and staying active. Then, re-test in 4 to 6 weeks.

If the levels remain abnormal after that, further diagnostic testing will be necessary to find the root cause.

As for M54, there isn't much more I can add here. We have a dedicated thread regarding referred pain, so feel free to ask for specifics over at Spinal and Back Pain.

Best, 🙂
goldencyclist372 said:I've had kidney infections three times now, and I've been fighting E. coli for quite a while. I take antibiotics for a bit, they clear it up, then it just comes right back. It’s a constant cycle. I'm not on any medication at the moment.
A few months ago, I had a kidney ultrasound and everything looked fine. I've done a urine culture and am currently waiting on those results.
Is it possible that this fresh blood suggests the presence of sand or kidney stones?
I've been dealing with a constant, dull ache in my kidneys for a couple of days.

Ma'am,

Given the presence of dysmorphic erythrocytes in your urine (cells that aren't shaped normally, which usually points toward a renal origin), my suggestion is this: once you have cleared the infection, you should definitely repeat the urine sediment test. If those abnormal cells show up again, you'll need a urine cytology performed three times and a consultation with a nephrologist.

It is certainly possible that the blood is caused by stones or "sand" in the kidneys—especially if there's a severe bladder infection—but the dysmorphic erythrocytes are the key detail to watch.

Best regards. 🙂
hollowraven54 said:Respectfully, I would appreciate some insight. My doctors haven't given me much to go on for two months now. I'm a woman in my early twenties. I went to my primary care physician because of wheezing in my chest, so she ordered blood work. These were the results: WBC 6.2 (ref: 3.4-9.7)
RBC: 4.35 (ref: 3.86-5.07)
Hemoglobin 133 (ref: 119-157)
Hematocrit 0.407 (ref: 0.365-0.47)
MCV: 93.4 (ref: 83-97.2)
MCH: 30.6 (ref: 27.4-33.9)
RDW-CV: 11.8 L (ref: 14.6-16.5)
Platelets: 293 (ref: 158-424)
MPV: 7.0 (ref: 6.8-10.4)
CBC
Neutrophils: 39.1 L (ref: 44-72)
Lymphocytes: 42.3 (ref: 20-46)
Monocytes: 9.1 (ref: 2-12)
Eosinophils: 8.4 H (ref: 0-7)
Basophils: 1.1 H (ref: 0-1)
After that, my doctor referred me to a pulmonologist where I underwent: inhaled allergy testing—negative; FeNo test—negative; IgE was 813 H (ref: div>

Ma'am,

Are the coughing and wheezing episodes still happening?

If they are, based on what you've described, you haven't been adequately evaluated for asthma. You haven't mentioned spirometry results, a bronchodilator response test (Albuterol challenge), or perhaps bronchoprovocation tests. In terms of diagnosing asthma, those carry far more weight than a FeNO test or a nasal swab for eosinophils.

For intermittent asthma (where symptoms are relatively mild and occur less than once a week), using Salbutamol (Albuterol) during flare-ups or for prevention is sufficient. However, one cannot make a definitive call on that without spirometry, a bronchodilator test, and a full assessment of pulmonary function.

Best regards. 🙂
stormytinker4 said:It’s not about what people say or hearsay; there are documented results available quite easily if you look online..

In that case, I’ll need links to the specific scientific studies confirming this efficacy. Otherwise, I’ll have to view this post as a violation of the Health subforum rules, which clearly state: "The Health subforum is strictly tied to official (conventional) medicine, grounded in science."