I want to apologize in advance because I know I’m basically about to dump an entire book on you all here!
It honestly feels like I’m some sort of blend of Woodie Allen-Jeremija...
I’ve decided to upload all my lab results from back in 1990 just to get them out there. I’m doing this because I’m honestly exhausted by the way doctors handle checkups lately; it feels like nobody actually looks at the big picture anymore. Everyone just stays in their own little lane and ignores how everything connects. Take my last neurologist, for example—I saw her twice, and frankly, I’m never stepping foot in her office again while I hunt for a new specialist. She tried to tell me that my vision issues were nothing more than a simple need for a new prescription. When I asked her about my VEP results, she basically brushed me off. This was frustrating because I had just seen an ophthalmologist right before that appointment, and I already had a fresh prescription and new glasses. My vision has never been perfect, but even with my current glasses, I see things clearly enough. If it were just a matter of needing a better prescription, I wouldn't be having these specific issues. Besides, my last VEP test was somewhere around 2003 or 2004, which means we're looking at an eight-year gap. It’s entirely possible things have worsened since then. But she just insisted that wasn't possible, arguing that without a recent VEP report showing conduction issues or optic nerve lesions, there was nothing to worry about. According to her, it had zero impact on my sight. She even made this sweeping claim that when people have optic nerve damage, they actually end up seeing better—which isn't describing my experience at all, but it's certainly an interesting take.
Back in 1989, I caught the Rubella, and things went south pretty quickly when I actually lost consciousness during the infection. About eight months later, out of nowhere, my sense of balance just completely vanished, and it took nearly a full year to start feeling even remotely normal again. It wasn't just being dizzy; it felt like I was constantly walking on ice while people were shoving me around from every direction, all while having this bizarre sensation that I’d been stuffed into a sack and was being tossed about. My vision was a mess, too—everything looked like I was looking through a tunnel or using binoculars, where I could only focus on what was right in front of me. At the time, my VEP results came back fine, and all the tests we ran seemed okay, though I never had a lumbar puncture or an MRI performed, even though the neurophysiologist strongly suggested them because of my symptoms and the fact that my abdominal reflexes were missing—she actually suspected it might be MS. Eventually, they explained to me that it was most likely post-infectious encephalomyelitis, but it makes you wonder how you're even supposed to go back and diagnose something like that after the fact.
Here are the diagnoses:
The diagnosis is an undifferentiated systemic connective tissue disease, specifically listed under code M35.9.
Antiphospholipid syndrome is one of those conditions that really keeps you on your toes because it’s so much more than just a simple diagnosis. It essentially means your immune system is acting a bit erratically, producing these antibodies that can trigger blood clots or complications during pregnancy. Dealing with it requires a lot of patience and a very steady hand from your medical team, but once you understand how it affects your body, you can start to manage it with a lot more confidence.
The patient is presenting with mild left-sided spastic hemiparesis.
There’s some suspicion that we might be looking at post-infectious encephalomyelitis disseminata.
Trigeminal neuralgia (G50.0) is something that really demands your full attention if you're dealing with it.
Mitral valve prolapse (I34.1)
Mitral valve insufficiency (I34.0) can be quite a heavy diagnosis to process when you first hear it, but it essentially means that one of the valves in your heart isn't closing quite as tightly as it should. Because the valve doesn't seal perfectly, some of the blood leaks backward instead of being pumped forward through the heart, which can eventually put extra strain on your system if it isn't managed properly. It’s one of those things where doctors usually want to keep a very close eye on how much leakage is actually happening to decide if you just need regular monitoring or if more active intervention is necessary.
The diagnosis is symptomatic epilepsy, specifically G40.4, though that label has actually been on the record since 2000, back when all the EEGs were being performed.
Everything was going perfectly fine, and then out of nowhere, I get hit with a diagnosis like that.
L5 nerve root radiculopathy on the right side along with bilateral S1 involvement, coded as G54.4.
Peripheral neuropathy in the lower extremities.
I've been dealing with some pretty intense cervical and brachial syndrome along with lumbar issues, specifically classified under M51.5.
Here’s what the rest of my results look like:
The upper and middle abdominal reflexes have been symmetrically absent since 1990, according to the most recent findings:
The KTR looks pretty weak on the left side during the middle stages, and honestly, everyone else just seems to be missing in action.
Back in 1990, I was dealing with platelet hyperaggregation.
The biopsy results came back showing lymphocytic hyperplasia, which basically means reactive lymphadenopathy—just a fancy way of saying the lymph nodes are reacting to something.
The EMNG results came back, and they show some chronic nerve damage in the muscles of the left lower leg as well as both sides.
Well, that's just great. My motor conduction velocity for the left peroneal nerve came back lower than it should be, and the right side is looking pretty similar.
The results show a symmetrical pattern characterized by prolonged distal latencies on both sides, which strongly suggests the presence of polyneuropathy.
I’ve been noticing some changes in my legs lately, specifically related to my diagnosis of L5 left-sided foraminal radiculopathy and bilateral sacroiliac joint issues.
The test results for my hands and feet show that I've got some moderate chronic radial nerve damage.
The distribution is hitting both sides of the L5 myotome, though it's more pronounced on the right, and the S1 myotome is also showing up bilaterally, just a bit more mild.
It looks like there's some bilateral involvement at the C7/C8 levels, specifically showing moderate chronic neural lesions within the muscles of the feet, though it’s more pronounced on the right side.
The analysis shows that both lists indicate a milder level of involvement within the hand muscles.
The Schirmer test results came back at 1mm for both eyes.
VEP: One finding shows that during stimulation of both eyes, the P100 wave amplitude is right on the borderline, accompanied by some prolonged latency.
The results show some latency issues, specifically indicating conduction delays in both pathways.
The results from both tests show that the P100 waves are registering with normal latency, though the amplitudes are coming in a bit lower than we typically like to see.
The results show that things are significantly lower on the left side compared to the right. Based on what the report is saying, the primary takeaway here points toward a neuronal lesion.
The left side looks fine, but the right side is coming back borderline.
The latest test results came back, and they show some neuronal lesions on both sides of the pathway, which might lead to some conduction issues or even more significant disruptions.
I’ve been thinking quite a bit lately about how strange it is when you start noticing a narrowing of your peripheral vision, almost like you're looking through a tunnel. It’s one of those subtle things that creeps up on you until you realize your field of view just isn't as wide as it used to be. If you're experiencing this, I really can't stress enough how important it is to get it checked out by an eye specialist right away, because you definitely don't want to ignore those kinds of visual changes.
The fourth result shows that when we stimulate the left eye, there’s a low-amplitude P100 wave present, though the latencies themselves look perfectly normal.
It’s really just about those upper quadrant fibers where you see that extended latency, which is exactly what we need to focus on if we're looking to trigger some effective stimulation.
The EEG shows some pretty clear P100 waves with steady amplitudes, though the latencies are definitely running a bit long. That’s what the findings show.
He’s talking about how there's some interference during both runs, specifically noting that it pulls significantly more to the right, and also regarding...
The scan showed a neuronal lesion in the lateral ventricular pathway.
The lumbar puncture results came back, and looking at the CSF protein electrophoresis, everything seems to be functioning normally aside from those elevated levels.
My prealbumin levels came back at 7.3, which seems to be a bit on the low side relative to the normal range.
BAER results basically point toward some kind of dysfunction within the auditory nerve pathway.
And his little nuggets, too.
When I'm sitting in the AG position, I’ve noticed that my left arm tends to oscillate and just sort of give out.
I noticed that the plantar reflexes seem to be weakened on the left side.
I've been dealing with some pretty intense diarrhea on my left side lately.
There's some noticeable loss of pigment on the tip of my nose.
In the Romberg position, there's noticeable instability when leaning back or toward the left.
The Romberg test—where you stand with your feet wide apart while holding onto something for balance—is a pretty straightforward way to check things out.
I've been dealing with some pretty intense trigeminal neuralgia lately, specifically targeting the areas covered by the second and third branches of the nerve on my right side.
The patient is presenting with some mild left-sided weakness, specifically a slight hemiparesis on the left side along with some facial drooping caused by a central type of seventh cranial nerve palsy on the left, while also experiencing decreased sensation, or hypesthesia, on that same side.
It looks like there might be some foreign objects more prominently visible on the leg.
The brain MRI came back looking good, though they did note a small, minor lesion.
Looking back at my neck MRI from 2006, the results showed a slight curve to the left, which they called a discrete left-convex scoliosis, along with some dorsal osteophytosis.
There is a circular zone of increased signal intensity located within the right area.
The MRI results show hyperintensities in the C3 and Th1 vertebral bodies on the T2-weighted images.
When you're looking at those T1-weighted MRI scans, seeing areas that show up as hypointense—basically, those darker spots on the image—is something we have to pay close attention to. It essentially means there's less signal coming from those specific tissues compared to the surrounding area, which can be caused by all sorts of things ranging from simple fluid buildup to more complex structural changes.
The X-ray results for the lumbar spine are back, showing dextroconvex scoliosis, hyperlordosis, and partial sacralization.
It looks like I'm dealing with some pseudoarthrosis, along with some subchondral sclerosis and narrowing in the L5-S1 disc space.
The imaging shows some subchondral sclerosis along the articular surfaces, along with some osteophyte formation in that specific zone.
It looks like we’re dealing with some bone density loss of unknown origin, specifically localized around the joints.
The imaging results show some sclerosis in the facet joints and there's a suspicion of spondylolysis, which basically means there might be a small stress fracture in the vertebrae. It looks like the issues are concentrated in the lower lumbar region.
The results regarding the joints show some concerning markers, so I’d definitely suggest looking into clinical correlation and scheduling some follow-up diagnostic testing to get a clearer picture, though nothing has been officially set in motion just yet.
I mean, nobody even suggested that it was necessary; they told me the results were perfectly fine, so why worry?
migraines
enteritis
My thyroid gland appears to be enlarged, though all of my hormone levels came back completely normal.
The echocardiogram results came back, and it looks like there is some mild telesystolic prolapse of the posterior mitral valve leaflet, along with a trace amount of mitral regurgitation.
It looks like we're looking at sinus tachycardia here.
The heart shows rhythmic activity, though you might notice it speeding up whenever there's a change in body position, and there is a mesosystolic murmur detected in the second intercostal space.
Since having my stroke, I’ve been dealing with these bouts of tachycardia that just seem to kick in out of nowhere, most of the time without any warning at all.
Whenever I change positions or try doing something like a few squats, my heart rate just spikes straight past 200 beats per minute.
Kidney biopsy results came back showing bilateral hydronephrosis.
Dynamic renal scintigraphy performed while sitting up shows that the right kidney has dropped down quite a bit.
I’ve been looking at my entire data set and comparing it against that baseline study we discussed earlier.
When looking at the results in a lying position, the renogram curve for the right kidney indicates that...
It looks like there’s a bit of a delay in reaching the peak secretion levels, along with a slightly slower elimination process than what we typically expect to see.
When you start looking into the activities occurring within the renal pelvis and the collecting system, you really begin to see how much heavy lifting the kidneys are doing behind the scenes. It isn't just about simple filtration; there is a whole sequence of movements and pressure changes that ensure everything flows exactly where it needs to go. You have to account for the way the urine moves through those various channels, following the structural layout of the renal pelvis to make sure the entire process stays efficient and steady.
- low potassium levels
- EEG results have been inconsistent, swinging between normal readings and occasional abnormalities, such as those dysrhythmic patterns
appearing in the left temporoparietal region and subparietal areas
- I've dealt with all sorts of skin issues on my face, ranging from eczema and rosacea to fungal infections
- My creatinine clearance is running a bit low, sitting somewhere around the 68-72 range